MaxATP Ingredients

 So, what are the ingredients in MaxATP?  Unlike many store bought “energy products” MaxATP is not a sugar/caffeine cocktail. Instead Max-ATP is a combination of vitamins, minerals and leading edge natural nutrition supplements to support and replenish your body’s healthy production of ATP.

 

Those ingredients are:

  • Ribose (a natural sugar you body must have to produce ATP)
  • N-acetyl- L-carnitine (which transports long chain fatty acids from the liver to be metabolized by the mitochondria)
  • RiboCeine – a Ribose-Cysteine compound which not only fuels the production of ATP but also supports the production of Glutathione (more)
  • Green Tea Extract – which provides antioxidant support and a NATURAL source of caffeine to boost your metabolism and promote mental alertness. Along with RiboCeine the green tea extract in MaxATP supports your production of Glutathione
  • Rhodiola rosea

    Rhodiola

    Rhodiola Rosea – to enhance mental performance and lessen the symptoms of fatigue. You might actually recognize this plant . Looks a lot like one of my garden favourites

  • Quercetin – an antioxidant
  • CoEnzyme Q10  another powerful antioxidant that stays in the mitochondria and supports both energy production and the elimination of toxic wastes
  • B Vitamins The B Vitamins act as coenzymes to help your body break down fats, amino acids and carbohydrates to produce energy. 
  • Magnesium is an essential catalyst in all energy dependent reactions in the body that include the use of ATP
  • Copper supports the synthesis of ATP in the mitochondria which is the part of the cell responsible for energy production
  • Chromium is not produced in the body and must come from diet -which is what the term essential means in relation to nutrient requirements. It helps cells absorb glucose (sugar)  and release energy.

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What is ATP?

ATP (adenosine triphosphate) is a naturally produced organic compound that’s made up of three different phosphate groups and adenosine (which includes ribose- a sugar). ATP is an abundant source of chemical energy which is stored in the phospate bound in it. When these bonds are broken the energy is released to become what is described and the currency that funds your metabolisms. ATP is produced in the mitochondria over every human cell via celluar respiration. Plants produce ATP in the chloroplasts through the process – photosynthesis.

As long as lots of ATP is present in your cells, your energy levels can be sustained and your cellular tissues will function properly. However, as with so many other things, as we age, our bodies become much less efficient at producing ATP. Our energy levels begin to decline and our cells do not function as effectively. It’s also useful to note, that while ATP stokes the fires of our metabolic processes at the cellular level, it also produces some waste products (just like the ashes in a fireplace) which left alone will accumulate in the cell and actually damage the mitochondria which produce it.

So, the process which produces our energy contains within it, the seeds of it’s own destruction. It’s all part of aging which may be inevitable, but there’s no reason to accept it prematurely. One substance that can help protect cellular mitochondria, from the waste it produces, is Glutathione, the body’s master antioxidant.

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RiboCeine Peer-Reviewed Research Summary

NOTE: The following links take you away from this page to 3rd party research sites.
1. Jurkowska, H.; Uchacz, T.; Roberts, J.; Wrobel, M. Potential therapeutic advantage of ribose-cysteine in the inhibition of astrocytoma cell proliferation Amino Acids, 2 April 2010

 

2. Walker, R.B.; Everette J.D., Comparative Reaction Rates of Various Antioxidants with ABTS Radical Cation. J. Agric Food Chem, 2009,57, 1156-1161.

 

3. Heman-Ackah, S.E.; Juhn, S.K.; Huang, T.C.; Wiedmann, T.S. Potential therapeutic advantage of ribose-cysteine in the inhibition of astrocytoma cell proliferation. Otolaryngology-Head and Neck Surgery, 2010, 143, 429-434.

 

4. Oz, H.S.; Chen, T.S.; Nagasawa, H., Comparative efficacies of 2 cysteine prodrugs and a glutathione delivery agent in a colitis model.Translational Research, 2007, 150(2), 122-129.

 

5. Lucas Slitt, A.M.; Dominick, P.K.; Roberts, J.C.; Cohen, S.D. Effect of Ribose Cysteine Pretreatment on Hepatic and Renal Acetaminophen Metabolite Formation and Glutathione Depletion. Basic Clin. Pharmacol. Toxicol., 2005, 96 (6), 487-94.

 

6. Lenarczyk, M.; Ueno, A.; Vannais, D.B.; Kraemer, S.; Kronenberg, A.; Roberts, J.C.; Tatsumi, K.; Hei, T.K.; Waldren, C.A. The “Pro-drug” RibCys Decreases the Mutagenicity of High-LET Radiation in Cultured Mammalian Cells. Radiation Research, 2003, 160, 579-583.

 

7. Wilmore, B.H.; Cassidy, P.B.; Warters, R.L.; Roberts, J.C. Thiazolidine Prodrugs as Protective Agents against y-Radiation-Induced Toxicity and Mutagenesis in V79 Cells. J. Med. Chem., 2001, 44(16), 2661-2666.

 

8. Lucus, A.M.; Henning G.; Dominick, P.K.; Whiteley, H.E.; Roberts, J.C.; Cohen, S.D. Ribose Cysteine Protects Against Acetaminophen-Induced Hepatic and Renal Toxicity. Toxicologic Pathology, 2000, 28(5), 697-704.

 

9.Roberts, J.C.; Phaneuf, H.L.; Dominick, P.K.; Wilmore, B.H.; Cassidy, P.B. Biodistribution of [35S] – Cysteine and Cysteine Prodrugs: Potential Impact on Chemoprotection Strategies. J. Labelled Cpd. Radiopharm., 1999, 42, 485-495.

 

10. Roberts, J.C.; Phaneuf, H.L.; Szakacs, J.G.; Zera, R.T.; Lamb, J.G.; Franklin, M.R. Differential Chemoprotection against Acetaminophen-Induced Hepatotoxicity by Latentiated L-Cysteines. Chem. Res. Toxicol., 1998, 11, 1274-1282.

 

11. Bantseev, V.; Bhardwaj, R.; Rathbun, W.; Nagasawa, H.T.; Trevithick, J.R. Antioxidants and Cataract: (Cataract Induction in Space Environment and Application to Terrestrial Aging Cataract). Biochem. Mol. Bio. Intl., 1997, 42, 1189-1197.

 

12. Roberts, J.C.; Koch, K.E.; Detrick, S.R.; Warters, R.L.; Lubec G. Thiazolidine Prodrugs of Cysteamine and Cysteine as Radioprotective Agents. Radiation Research, 1995, 143, 203-213.

 

13. Carroll, M.P.; Zera, R.T.; Roberts, J.C.; Schlafmann, S.E.; Feeny, D.A.; Johnston, G.R.; West, M.A.; Bubrick, M.P. Efficacy of Radioprotective Agents in Preventing Small and Large Bowel Radiation Injury. Dis. Colon Rectum, 1995, 38(7), 716-722.

 

14. Roberts, J.C.;Francetic, D.J.; Zera, R.T. Chemoprotection against Cyclophosphamide-Induced Urotoxicity: Comparison of Nine Thiol Protective Agents. AntiCancer Research, 1994, 14, 389-396.

 

15. Rowe, J.K.; Zera, R.T.; Madoff, R.D.; Fink, A.S.; Roberts, J.C.; Johnston, G.R.; Freeney, D.A.;Young, H.L.; Bubrick, M.P. Protective Effect of RibCys Following High-Dose Irradiation of the Rectosigmoid. Dis. Colon Rectum, 1993, 36(7), 681-687.

 

16. Roberts, J.C.; Charyulu, R. L.; Zera, R.T.; Nagasawa, H.T. Protection Against Acetaminophen Hepatotoxicity by Ribose-Cysteine (RibCys). Pharmacology & Toxicology, 1992, 70, 281-285.

 

17. Roberts, J.C.; Francetic, D.J. – Mechanisms of Chemoprotection by RibCys, a Thiazolidine Prodrug of L-cysteine. Med. Chem. Res.,1991, 1, 213-219.

 

18. Roberts, J.C.; Francetic, D.J. Time course for the elevation of glutathione in numerous organs of L1210-bearing CDF1 mice given the L-cysteine prodrug, RibCys. Toxicology Letters, 1991, 59, 245-251.

 

19. Roberts, J.C.; Francetic, D.J.; Zera, R.T. L-cysteine prodrug protects against cyclophosphamide urotoxicity without compromising therapeutic activity. Cancer Chemotherapy and Pharmacology 1991, 28, 166-170.

 

20. Roberts, J.C.; Nagasawa, H.T.; Zera, R.T.; Fricke, R.F.; Goon, D.J. W. Prodrugs of L-cysteine as protective agents against acetaminophen-induced hepatotoxicity. 2-(polyhydroxyalky)-and 2-(Polyacetoxyalky)-Thiazolidine-4(R)-Carboxylic Acids. J. Med Chem.1987, 30, 1891-1896.

 

Save on MaxGXL, MaxOne, MaxATP & Max N-FUZE – The Max International Loyalty Program

For both new and already loyal users of MaxGXL, Max GXL Sport, MaxONE, MaxATP and Max N-FUZE and Cellgevity, Max International has introduced a Customer Loyalty Program that will add convenience and save you money.  In fact, considering that you can save $19.00 a box on MaxGXL and Max GXL Sport and $15.00 off a one month supply of MAXONE, Max N-FUZE  or a half pack of Max ATP, there are some terrific savings to be had on this program.

The offer is simple – Sign up for an autoship and commit to stay on it for at least three months.  In exchange for your commitment to invest in your health for the next three months – you get an incredible deal.  At the end of three months, you will have lived up to your end of the bargain.  After that, should you choose, you can cancel anytime without penalty. If you cancel within the first three months however, there is a cancellation fee of $60.00 for each product unit you have on the loyalty program.

Signing up for the Loyalty program is simple.  Call 1-866-993-6243.  We’ll take your information and get you started.  If you’re already an autoship customer, talk to your distributor or call Max International about the Loyalty Program.